Key Product Details
Description: | CaV2.2 blocker |
Purity: | ≥95% (HPLC) |
Molecular Weight: | 3037 |
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Citations for ω-Conotoxin GVIA (15)
Citations are publications that use Bio-Techne products. Selected citations for ω-Conotoxin GVIA include:
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Bellono
(2017)
Molecular basis of ancestral vertebrate electroreception.
Nature.
543:391
PMID: 28264196 -
Cassilly et al.
(2016)
SB-224289 Antagonizes the Antifungal Mechanism of the Marine Depsipeptide Papuamide A.
Front Cell Neurosci.
11:e0154932
PMID: 27183222 -
Perissinotti et al.
(2015)
Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice.
Proc Natl Acad Sci U S A.
8:444
PMID: 25610372 -
Pan et al.
(2014)
σ-1 receptor antagonism restores injury-induced decrease of voltage-gated Ca2+ current in sensory neurons.
Addict Biol.
350:290
PMID: 24891452 -
Mahler et al.
(2014)
Modafinil attenuates reinstatement of cocaine seeking: role for cystine-glutamate exchange and metabotropic glutamate receptors.
J Neurosci.
19:49
PMID: 23017017 -
Szabó et al.
(2014)
Presynaptic calcium channel inhibition underlies CB1� cannabinoid receptor-mediated suppression of GABA release.
PLoS One.
34:7958
PMID: 24899717 -
Medrihan et al.
(2013)
Synapsin II desynchronizes neurotransmitter release at inhibitory synapses by interacting with presynaptic calcium channels.
Nature.
4:1512
PMID: 23443540 -
Schuwald et al.
(2013)
Lavender oil-potent anxiolytic properties via modulating voltage dependent calcium channels.
J Neurosci.
8:e59998
PMID: 23637742 -
Smith et al.
(2013)
Comparative functional expression of nAChR subtypes in rodent DRG neurons.
Front Cell Neurosci.
7:225
PMID: 24348328 -
Izzo et al.
(2012)
Inhibitory effect of cannabichromene, a major non-psychotropic cannabinoid extracted from Cannabis sativa, on inflammation-induced hypermotility in mice.
Br J Pharmacol.
166:1444
PMID: 22300105 -
Kim and Alger
(2010)
Reduction in endocannabinoid tone is a homeostatic mechanism for specific inhibitory synapses.
Nat Neurosci.
13:592
PMID: 20348918 -
Grubb and Burrone
(2010)
Activity-dependent relocation of the axon initial segment fine-tunes neuronal excitability.
J Pharmacol Exp Ther.
465:1070
PMID: 20543823 -
Chang
(2010)
Dynamic modulation of phasic and asynchronous glutamate release in hippocampal synapses.
J Neurophysiol.
103:392
PMID: 19889850 -
Chu et al.
(2009)
Secr. as a neurohypophysial factor regulating body water homeostasis.
PLoS One.
106:15961
PMID: 19805236 -
Haeberle et al.
(2004)
Molecular profiling reveals synaptic release machinery in Merkel cells.
Proc Natl Acad Sci U S A.
101:14503
PMID: 15448211
There are no citations that match your criteria.
Customer Reviews for ω-Conotoxin GVIA (3)
Customer Images
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Species: OtherAssay Type: In VitroThe following concentrations we were used:1 μM Bay K, 10 μM nifedipine, 10 μM nimodipine, 300 nM omega -agatoxin, 1 μM omega -conotoxin, 5 μM mibefradil, 1 μM tetrodotoxin, 1 μM charybdotoxin, 100 nM iberiotoxin, 10 μM NS11021.
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Species: OtherAssay Type: In VitroBiologically active compounds that have been documented to interact with awide range of targets including SB-224289 (Cat. # 1221), MG-624 (Cat.# 1356), GVIA (Cat. No. 1085), DPAT hydrobromide (Cat. No. 1080) and valinomycin (VA; Cat. # 3373) were ordered from Tocris Bioscience
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Species: RatAssay Type: Ex VivoCell Line/Tissue: Anterior cingulate cortexExperiment to test dependence of field EPSPs in neural tissue from N-type Ca2+ channels. 10 min incubation with 25 microM omega -Conotoxin GVIA decreased significantly fEPSP amplitude. Blue: control. Red: after incubation with omega -Conotoxin GVIA.
There are no reviews that match your criteria.