ω-Conotoxin GVIA

Citations are publications that use Bio-Techne products. Selected citations for ω-Conotoxin GVIA include:

• Bellono (2017) Molecular basis of ancestral vertebrate electroreception. Nature. 543:391
  PMID: 28264196
• Cassilly et al. (2016) SB-224289 Antagonizes the Antifungal Mechanism of the Marine Depsipeptide Papuamide A. Front Cell Neurosci. 11:e0154932
  PMID: 27183222
• Perissinotti et al. (2015) Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice. Proc Natl Acad Sci U S A. 8:444
  PMID: 25610372
• Pan et al. (2014) σ-1 receptor antagonism restores injury-induced decrease of voltage-gated Ca2+ current in sensory neurons. Addict Biol. 350:290
  PMID: 24891452
• Mahler et al. (2014) Modafinil attenuates reinstatement of cocaine seeking: role for cystine-glutamate exchange and metabotropic glutamate receptors. J Neurosci. 19:49
  PMID: 23017017
• Szabó et al. (2014) Presynaptic calcium channel inhibition underlies CB1� cannabinoid receptor-mediated suppression of GABA release. PLoS One. 34:7958
  PMID: 24899717
• Medrihan et al. (2013) Synapsin II desynchronizes neurotransmitter release at inhibitory synapses by interacting with presynaptic calcium channels. Nature. 4:1512
  PMID: 23443540
• Schuwald et al. (2013) Lavender oil-potent anxiolytic properties via modulating voltage dependent calcium channels. J Neurosci. 8:e59998
  PMID: 23637742
• Smith et al. (2013) Comparative functional expression of nAChR subtypes in rodent DRG neurons. Front Cell Neurosci. 7:225
  PMID: 24348328
• Izzo et al. (2012) Inhibitory effect of cannabichromene, a major non-psychotropic cannabinoid extracted from Cannabis sativa, on inflammation-induced hypermotility in mice. Br J Pharmacol. 166:1444
  PMID: 22300105
• Kim and Alger (2010) Reduction in endocannabinoid tone is a homeostatic mechanism for specific inhibitory synapses. Nat Neurosci. 13:592
  PMID: 20348918
• Grubb and Burrone (2010) Activity-dependent relocation of the axon initial segment fine-tunes neuronal excitability. J Pharmacol Exp Ther. 465:1070
  PMID: 20543823
• Chang (2010) Dynamic modulation of phasic and asynchronous glutamate release in hippocampal synapses. J Neurophysiol. 103:392
  PMID: 19889850
• Chu et al. (2009) Secr. as a neurohypophysial factor regulating body water homeostasis. PLoS One. 106:15961
  PMID: 19805236
• Haeberle et al. (2004) Molecular profiling reveals synaptic release machinery in Merkel cells. Proc Natl Acad Sci U S A. 101:14503
  PMID: 15448211

There are no citations that match your criteria.

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• The pharmacological inhibitors for electrophysiology assay

  Name: Anonymous
  Species: Other
  Assay Type: In Vitro
  The following concentrations we were used:1 μM Bay K, 10 μM nifedipine, 10 μM nimodipine, 300 nM omega -agatoxin, 1 μM omega -conotoxin, 5 μM mibefradil, 1 μM tetrodotoxin, 1 μM charybdotoxin, 100 nM iberiotoxin, 10 μM NS11021.
  PMID: 28264196
  

• The 5,760 bioactive compound library is a collated compound set of approved drugs and biologically active compound

  Name: Anonymous
  Species: Other
  Assay Type: In Vitro
  Biologically active compounds that have been documented to interact with awide range of targets including SB-224289 (Cat. # 1221), MG-624 (Cat.# 1356), GVIA (Cat. No. 1085), DPAT hydrobromide (Cat. No. 1080) and valinomycin (VA; Cat. # 3373) were ordered from Tocris Bioscience
  PMID: 27183222
  

• omega -Conotoxin suppresses field EPSP amplitude

  Name: Anonymous
  Species: Rat
  Assay Type: Ex Vivo
  Cell Line/Tissue: Anterior cingulate cortex
  Experiment to test dependence of field EPSPs in neural tissue from N-type Ca2+ channels. 10 min incubation with 25 microM omega -Conotoxin GVIA decreased significantly fEPSP amplitude. Blue: control. Red: after incubation with omega -Conotoxin GVIA.
  PMID: 17208660
  

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