(S)-(-)-Sulpiride

Citations are publications that use Bio-Techne products. Selected citations for (S)-(-)-Sulpiride include:

• Pedersen et al. (2021) A novel luminescence-based-β-arrestin recruitment assay for unmodified receptors. J Biol Chem. 2021;
  PMID: 33684444
• Dorst et al. (2020) Polysynaptic inhibition between striatal cholinergic interneurons shapes their network activity patterns in a dopamine-dependent manner. Nat Commun. 11:5113
  PMID: 33037215
• Beas et al. (2018) The locus coeruleus drives disinhibition in the midline thalamus via a dopaminergic mechanism. Nat Neurosci. 21:963
  PMID: 29915192
• Dai et al. (2018) Selective blockade of spinal D2DR by levo-corydalmine attenuates MOR tolerance via suppressing PI3K/Akt-MAPK signaling in a MOR-dependent manner. Exp Mol Med. 50:148
  PMID: 30429454
• Shin et al. (2015) Muscarinic regulation of DA and glutamate transmission in the nucleus accumbens. J Clin Invest. 112:8124
  PMID: 26080439
• Kim et al. (2015) Spinal DArgic projections control the transition to pathological pain plasticity via a D1/D5-mediated mechanism. J Neurophysiol. 35:6307
  PMID: 25904784
• Ma et al. (2015) Agonist and antagonist effects of aripipr. on D₂-like receptors controlling rat brain DA synthesis depend on the DArgic tone. Int J Neuropsychopharmacol. 18
  PMID: 25522390
• Jensen (2015) Effects of DA D2-Like Receptor Antagonists on Light Responses of Ganglion Cells in Wild-Type and P23H Rat Retinas. PLoS One. 10:e0146154
  PMID: 26717015
• Brzosko et al. (2015) Retroactive modulation of spike timing-dependent plasticity by DA. Elife. 4
  PMID: 26516682
• Marcott et al. (2014) Phasic DA release drives rapid activation of striatal D2-receptors. Neuron. 84:164
  PMID: 25242218
• Dragicevic et al. (2014) Cav1.3 channels control D2-autoreceptor responses via NCS-1 in substantia nigra DA neurons. J Neurosci. 137:2287
  PMID: 24934288
• Cilz et al. (2014) DArgic modulation of GABAergic transmission in the entorhinal cortex: concerted roles of α1 adrenoreceptors, inward rectifier K⁺, and T-type Ca²⁺ channels. Cereb Cortex. 24:3195
  PMID: 23843440
• Lim et al. (2014) A leptin-mediated central mechanism in analgesia-enhanced opioid reward in rats. J Neurosci. 34:9779
  PMID: 25031415
• Baca et al. (2013) Gene-environment interactions affect long-term depression (LTD) through changes in DA receptor affinity in Snap25 deficient mice. Brain Res. 1532:85
  PMID: 23939223
• Ishima et al. (2012) Neurite outgrowth mediated by the heat shock protein Hsp90α: a novel target for the antipsychotic drug aripipr. Transl Psychiatry. 2:e170
  PMID: 23047241
• Kurita et al. (2012) HDAC2 regulates atypical antipsychotic responses through the modulation of mGlu2 promoter activity. Nat Neurosci. 15:1245
  PMID: 22864611
• Chuhma et al. (2009) DA neuron glutamate cotransmission: frequency-dependent modulation in the mesoventromedial projection. Neuroscience. 164:1068
  PMID: 19729052
• Costa et al. (2008) Electrophysiology and pharmacology of striatal neuronal dysfunction induced by mitochondrial complex I inhibition. J Neurosci. 28:8040
  PMID: 18685029
• Maher and Westbrook (2008) Co-transmission of DA and GABA in periglomerular cells. Proc Natl Acad Sci U S A. 99:1559
  PMID: 18216231
• Wang et al. (2006) DArgic control of corticostriatal long-term synaptic depression in medium spiny neurons is mediated by cholinergic interneurons. Neuron. 50:443
  PMID: 16675398
• Yang et al. (2004) Inhibition of ATP-sensitive potassium channels by halope. Br J Pharmacol. 143:960
  PMID: 15533888

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• Selective block of dopamine receptors

  Name: Sergiy Sylantyev
  Species: Human
  Cell Line/Tissue: Stem cell - derived neurons
  (S)‐sulpiride (Sul) was in use to block D2 dopamine receptors and thus prevent D2-mediated suppression of NMDA receptors response. The compound worked as expected, generating a discernible increase of amplitudes of NMDA-receptor – mediated evoked post-synaptic potentials in cultured dopamine neurons when applied with SCH‐23390 (see illustration). No problem with dissolution in DMSO for 10 mM stock.
  Reference
  

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